Over-expression of parvalbumin in transgenic mice rescues motoneurons from injury-induced cell death
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Dekkers, J.
Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology, London UK
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Bayley, P.
Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology, London UK
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Dick, J. R. T.
Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology, London UK
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Schwaller, Beat
Institute of Histology, Department of Medicine, University of Fribourg, Switzerland
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Berchtold, M. W.
Department of Molecular Cell Biology, Institute of Molecular Biology, University of Copenhagen, Denmark
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Greensmith, L.
Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology, London UK
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Published in:
- Neuroscience. - 2004, vol. 123(2), no. 459-466
English
Following nerve injury in neonatal rats, a large proportion of motoneurons die, possibly as a consequence of an increase in vulnerability to the excitotoxic effects of glutamate. Calcium-dependent glutamate excitotoxicity is thought to play a significant role not only in injury-induced motoneuron death, but also in motoneuron degeneration in diseases such as amyotrophic lateral sclerosis... Show more…
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Faculty
- Faculté des sciences et de médecine
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Department
- Département de Médecine
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Language
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Classification
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Biology
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License
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License undefined
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Identifiers
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Persistent URL
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https://folia.unifr.ch/unifr/documents/299544