Journal article

Aldosterone-induced serum and glucocorticoid-induced kinase 1 expression is accompanied by Nedd4-2 phosphorylation and increased Na+ transport in cortical collecting duct cells

  • Flores, Sandra Y. Department of Pharmacology and Toxicology, CHUV, University of Lausanne, Switzerland
  • Loffing-Cueni, Dominique Department of Pharmacology and Toxicology, CHUV, University of Lausanne, Switzerland
  • Kamynina, Elena Department of Pharmacology and Toxicology, CHUV, University of Lausanne, Switzerland
  • Daidié, Dorothée Department of Pharmacology and Toxicology, CHUV, University of Lausanne, Switzerland
  • Gerbex, Carole Department of Pharmacology and Toxicology, CHUV, University of Lausanne, Switzerland
  • Chabanel, Sting Department of Pharmacology and Toxicology, CHUV, University of Lausanne, Switzerland
  • Dudler, Jean Department of Rheumatology, CHUV, University of Lausanne, Switzerland
  • Loffing, Johannes Department of Pharmacology and Toxicology, CHUV, University of Lausanne, Switzerland - Department of Medicine, Unit of Anatomy, University of Fribourg, Switzerland
  • Staub, Olivier Department of Pharmacology and Toxicology, CHUV, University of Lausanne, Switzerland
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    04.05.2005
Published in:
  • Journal of the American Society of Nephrology. - 2005, vol. 16, no. 2279
English Aldosterone plays a central role in Na⁺ homeostasis by controlling Na⁺ reabsorption in the aldosterone-sensitive distal nephron involving the epithelial Na⁺ channel (ENaC). Part of the effects of aldosterone is mediated by serum and glucocorticoid-induced kinase 1 (Sgk1), a Ser/Thr kinase whose expression is rapidly induced by aldosterone and that increases in heterologous expression systems... Show more…
Faculty
Faculté des sciences et de médecine
Department
Département de Médecine
Language
  • English
Classification
Biology
License
License undefined
Identifiers
Persistent URL
https://folia.unifr.ch/unifr/documents/299863