Nitric oxide synthase gene transfer inhibits biological features of bypass graft disease in the human saphenous vein
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Tanner, Felix C.
Cardiovascular Research, Clinic for Cardiovascular Surgery and Department of Clinical Research, University Hospital, Bern, Switzerland
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Largiadèr, Thomas
Cardiovascular Research, Physiology Institute, University Zürich-Irchel and Division of Cardiology, University Hospital, Zürich, Switzerland
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Greutert, Helen
Cardiovascular Research, Clinic for Cardiovascular Surgery and Department of Clinical Research, University Hospital, Bern, Switzerland
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Yang, Zhihong
Physiology Institute, University of Fribourg, Fribourg, Switzerland
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Lüscher, Thomas F.
Cardiovascular Research, Physiology Institute, University Zürich-Irchel and Division of Cardiology, University Hospital, Zürich, Switzerland
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Published in:
- Journal of Thoracic and Cardiovascular Surgery. - 2004, vol. 127, no. 1, p. 20-26
English
Background Bypass graft disease is related to proliferation and migration of vascular smooth muscle cells and to platelet activation with thrombus formation. Nitric oxide inhibits these biological responses; it has never been demonstrated, however, whether this occurs in intact human vascular tissue after endothelial nitric oxide synthase gene transfer. Methods We examined whether endothelial... Show more…
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Faculty
- Faculté des sciences et de médecine
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Department
- Département de Médecine
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Language
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Classification
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Medicine
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License
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License undefined
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Identifiers
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Persistent URL
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https://folia.unifr.ch/unifr/documents/299568